Why would a human cell that maintains an antiviral state to long will die?

Amely Wyman asked a question: Why would a human cell that maintains an antiviral state to long will die?
Asked By: Amely Wyman
Date created: Mon, Aug 16, 2021 6:37 AM

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❓ A human cell that maintains an antiviral state of blood?

A human cell that maintains an antiviral state induced by alpha or beta interferons for too long will die because _____. it cannot make proteins Neutrophils can kill bacteria by nonphagocytic mechanisms.

❓ A human cell that maintains an antiviral state of emergency?

A human cell that maintains an antiviral state induced by alpha or beta interferons for too long will die because _____. it cannot engage in glucose metabolism it cannot move it cannot repair its cytoplasmic membrane it cannot make proteins

❓ A human cell that maintains an antiviral state of water?

Why would a human cell that maintains an antiviral state to long will die? Wiki User. ∙ 2014-12-09 18:00:44. Want this question answered? Be notified when an answer is posted.

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Why would a human cell that maintains an antiviral state to long will die? Wiki User. ∙ 2014-12-09 18:00:44… Why would a human cell that maintains an antiviral state to long will die?

A human cell that maintains an antiviral state induced by alpha or beta interferons for too long will die because _____.-it cannot engage in glucose metabolism-it cannot make proteins-it cannot repair its cytoplasmic membrane-it cannot move

The antiviral state results in the cessation of protein synthesis in a cell, so if this is prolonged, the cell dies.

The cell-mediated response occurs when the lymphokines released from the helper T-cells stimulate other cell types to participate in the immune response. Lymphokine-stimulated killer T-cells attach to the pathogen-infected cells and destroy them, whereas lymphokine-activated phagocytic cells produce more toxic molecules that can kill the pathogen directly.

Interferon-β (IFNβ) induces strong antiviral effects and is therefore an attractive agent to prevent or reduce the incidence of virus-mediated exacerb…

Intracellular human gamma-interferon triggers an antiviral state in transformed murine L cells. Proceedings of the National Academy of Sciences, 1987. Catherine Vaquero. Florence Béranger. F. Beranger. Josiane Sancéau. Catherine Vaquero. Florence Béranger.

TLR-9 Contributes to the Antiviral Innate Immune Sensing of Rodent Parvoviruses MVMp and H-1PV by Normal Human Immune Cells Zahari Raykov1, Svitlana P. Grekova1, Rita Ho¨rlein1, Barbara Leuchs1 ...

The HBV genome persists despite an immune response in the form of covalently closed circular DNA (cccDNA) and integrated DNA, rendering an occult state of viral persistence in individuals whose infection appears to have been resolved. We have described HBV reactivation that occurs because of changes in the virus or the immune system.

Indeed, why infected CD4+ T cells die when infected with HIV, while other cells such as microglia can be infected for years, likely depends on formation of this specialised IFI16 inflammasome . In addition to the activation of innate signalling pathways as we have seen above, IFI16 has other more direct antiviral activities, especially to restrict herpesviruses.

Author summary Influenza A virus (IAV) is a respiratory virus that can infect multiple cell types in the lung. It is not known how individual cell types contribute to the innate immune response. Additionally, it is not known how distinct stages of early IAV replication are detected by infected cells. To address these questions, we use a combination of viruses that can replicate but not spread ...

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